IÑIGO LASA UZCUDUN
Catedrático Universidad

• Department: CIENCIAS DE LA SALUD
• Area: Microbiología
• e-mail: ilasaunavarra.es
• Phone number: 848422486 (ext. )
•   NAVARRABIOMED

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• Additional information

Current position:   Director of Navarrabiomed, Center of Biomedical Research, Pamplona. Spain

• 1983-1988       B.Sc. Biology. University of Navarra. Spain
• 1992               PhD thesis in Science, Universidad Autonoma de Madrid.     
• 1995-1997       EU Postdoctoral fellow.Unité des interactions Bacteries-Celules. Institut Pasteur. France.
• 1998 – 2008     Assistant Professor of Microbiology. Universidad Pública de Navarra 
• 2008-present   Professor of Microbiology. Universidad Pública de Navarra.

• Editorial board of Molecular Microbiology (2008-2010)
• Head of the Management Board (MB) of the Biotechnology grant program of the Spanish Ministry
• Visiting scientist. Cold Spring Harbor Laboratory. USA

2001 “Ortiz de Landazuri” award. Gobierno de Navarra, Spain

2005 “Jaime Ferran” award. Sociedad Española de Microbiología. Cáceres. Spain

2006 Research award on Basic Science of the Public University of Navarra. Spain

·     Total articles in publication list: 102

·     Articles with citation data: 91

·     Sum of the times cited: 5750

·     Average citations per article: 60,55

·     h-index: 44

·     Last updated: 12/12/2018

Our laboratory investigates fundamental mechanisms of regulation of bacterial biofilm development with a focus on human pathogens. We are interested in understanding how signal transduction systems control the transition between planktonic and sessile lifestyles. We also study the composition of biofilm matrix and the function of these compounds during infection. In particular, we do research on basic principles of RNA overlapping transcription and its role in gene expression, on mechanisms of exopolysaccharide synthesis regulation and on how surface proteins of the biofilm matrix mediated intercellular adhesion and interactions with the host cells. Very recently, we have deconstructed the two-component signal transduction system of Staphylococcus aureus. In this study, we have generated fifteen S. aureusstrains, each containing just a single TCS, as well as a mutant lacking all 15 non-essential TCS. This powerful and unique genetic resource will allow us to unambiguously determine the contribution that each individual TCS to the regulation of peptidoglycan biosynthesis and antibiotic resistance.